Cerebrolysin 5ml × 10 Ampoules· 5ml × 10 ampoules

The proposed mechanism of action of cerebrolysin involves multi-targeted neurotrophic and neuroprotective effects mediated by the low-molecular-weight peptide fraction. The preparation has been reported to engage TrkB and TrkA receptor signaling in a manner reminiscent of endogenous BDNF and NGF, activating downstream CREB and ERK pathways that support neuronal survival and plasticity.

Additional mechanisms documented in preclinical studies include modulation of amyloid precursor protein processing, reduction of caspase-3 activation in models of neuronal injury, attenuation of glutamate excitotoxicity, and reduction of oxidative stress markers. In stroke models, the preparation has been associated with reductions in infarct volume and preservation of neurological function. In Alzheimer's disease preclinical models, effects on amyloid pathology and synaptic markers have been reported. The complex composition of the preparation precludes attribution of specific effects to single peptide species, and active component identification remains an area of continued investigation.